HYPERPROLACTINEMIA: GALACTORRHEA-AMENORRHEA SYNDROME - Etiology
Prolactin is under chronic inhibitory control by a hypothalamic factor (presumably dopamine). Any pharmacologic agent or lesion of the hypothalamus or pituitary stalk that interferes with dopamine secretion or its action can result in hy-perprolactinemia. In view of the wide clinical use of pharmacologic agents that interfere with dopaminergic transmission, such as the phenothia-zines, it is not surprising that the majority of patients with hyperprolactinemia do not harbor a pituitary adenoma. Once the use of such an agent can be excluded, however, and mechanical or neurogenic factors eliminated, the frequency with which a pituitary microadenoma is found is quite high. The most common pituitary tumor, chromophobe adenoma, once thought to be nonfunctioning and endocrinologically silent, is frequently a prolactin-secreting adenoma (prolactinoma).
- GIGANTISM AND ACROMEGALY
- PITUITARY NEOPLASMS: GENERAL CONSIDERATIONS
- GIGANTISM AND ACROMEGALY - Treatment
- HYPERPROLACTINEMIA: GALACTORRHEA-AMENORRHEA SYNDROME - Clinical Manifestations
- GIGANTISM AND ACROMEGALY - Diagnosis
- DISORDERS OF THE POSTERIOR PITUITARY
- HYPERPROLACTINEMIA: GALACTORRHEA-AMENORRHEA SYNDROME - Treatment
- HYPERPROLACTINEMIA: GALACTORRHEA-AMENORRHEA SYNDROME - Etiology
- HYPERPROLACTINEMIA: GALACTORRHEA-AMENORRHEA SYNDROME - Diagnosis
- SYNDROMES OF ANTERIOR PITUITARY HYPERFUNCTION: THE PITUITARY ADENOMA